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solving nonverbal items. (Three of the five WAIS-R Performance subtests loaded in the .50s on Verbal Comprehension for these middleaged men.) In addition, at ages 55 74, neither sex applied verbal strategies to WAIS-R Performance tasks to any marked degree. Thus, an overview of the WAIS-R factor structures for males and females attests to the similarity of these patterns. Where differences occur, they are as likely to disagree as to agree with predictions made on the basis of Inglis and Lawson s (1982) hypothesis. In fact, only one instance gives the Inglis/Lawson hypothesis good support: African American females, compared to African American males, a topic that is treated in the section on race differences in brain-damage studies. Correlation coefficients between WAIS-III V-IQ and P-IQ, separate by gender, are not available, and neither are separate factor analyses of the WAIS-III. Therefore, the WAIS-R data will have to suffice for the discussion of the Inglis/Lawson interpretation of gender differences among patients with lateralized lesions. However, as indicated previously, the pertinent neuropsychological investigations of unilateral brain damage on Wechsler s Verbal and Performance scales have been conducted with the WAIS-R and its predecessors, not with the WAIS-III. Turkheimer et al. s Evaluation of the McGlone Hypothesis versus the Inglis/ Lawson Hypothesis Turkheimer et al. (1993) recognized that the question remains unresolved concerning why gender differences occur in the effects of lateralized lesions. They noted that two of the major explanatory hypotheses are those we discussed: McGlone s lateralization hypothesis and Inglis and Lawson s differential problem-solving hypothesis. Turkheimer et al. (1993) analyzed the data of 33 males and 31 females using a complicated quantitative method for the assessment of covariation between IQ and location of the lesion involving model-fitting to the Cartesian

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coordinates of points in the patients CT scans to discern which hypothesis was supported The plausibility of the hypothesis that gender difference is largely the result of the degree of lateralization in males versus females (McGlone, 1977) was not supported by Turkheimer et al s results because a statistical model in which the genders have the same degree of lateralization fit the data as well as a model in which the genders were allowed to differ In contrast, Turkheimer et al found statistical support for Inglis and Lawson s (1982) hypothesis that females use verbal strategies in solving Performance IQ items A single model of lesion effects accounted for deficits in V-IQ and P-IQ in females, but not in males.

B.1.5

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The results suggested that left-hemisphere lateralposterior lesions caused the most severe deficits for male and female V-IQ, but the relationship between lesion location and P-IQ was considerably more complex for both genders Although support was found for Inglis and Lawson s (1982) hypothesis in Turkheimer et al s (1993) analysis of data based on a total of only 64 subjects, recall that summary data from Turkheimer and Farace s (1992) meta-analysis of 12 studies (Table 813) did not support predictions about V-IQ and P-IQ based on Inglis and Lawson s hypothesis (Table 815) In addition, Turkheimer et al (1993) posit that there are multiple possible reasons for the patterns of deficit, not just the problem-solving strategies that were emphasized by Inglis and Lawson Turkheimer et al note that the strategy, brain organization, or both, could account for the patterns found for males and females.

The most striking pattern that Turkheimer et al found was that left-lesioned females had substantial P-IQ deficits related to lesion parameters, but left-lesioned males did not Possible reasons for this discrepancy in gender patterns include the hypotheses that females have more nonverbal abilities relevant to P-IQ represented in the left hemisphere or that females use more verbal strategies in solving P-IQ items Because P-IQ comprises more than one cognitive ability, ambiguity arises about the reasons underlying these patterns..

Turkheimer et al. suggest that the best way to determine whether problem-solving strategies or localization of function is responsible for the gender effects is to decompose the P-IQ items into more basic cognitive tasks. Certainly the four factor indexes yielded by the WAIS-III help achieve that goal to some extent, and future gender-based WAIS-III neuropsychological research that features the index profile may help resolve the reason for gender differences in lateralized lesion studies.

Turkheimer et al. s (1993) analysis lent more support to the Inglis/Lawson hypothesis than to McGlone s hypothesis, but Turkheimer et al. also recognized that more complex studies with larger samples are necessary for clarification. The results of empirical analyses discussed in earlier sections of this chapter and shown in Tables 8.13, 8.16, 8.17, and 8.18 generally contradict the Inglis/ Lawson hypothesis and give more support to McGlone s (1977, 1978, 1980) notion of less asymmetry in the cerebral organization of female versus male brains. Anatomically, Witelson (1989) notes that right-handed males have greater cortical asymmetry than females in the temporal and parietal regions of the brain, and the part of the corpus callosum that connects these asymmetric regions is larger in females than males. Furthermore, the corpus callosum gets smaller for males with increasing age (between 25 and 68), but not for females (Lezak, 1995), which may make it less likely for older men than older women to use both sides of the brain to compensate for an injury to one hemisphere. One reason for the failure of researchers to consistently find gender differences in cerebral organization may be that the difference resides in the potential for females to compensate for damage or dysfunction. Perhaps women tend to solve problems in a manner similar to men under ordinary circumstances. However, when the cir-

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